Development of tinea and candidiasis
Tinea corporis is a fungi infection caused by dermatophytes. Dermatophyte is a mold-like fungus that affects dead tissues. Infection sites associated with tinea corporis or ringworm includes scalp, groin, toes, and the neck region. However, the prevalence of the disease is associated with children. The predisposing factors include clothing, comb, pool surface, showers and walls (Clayton & Noble, 2010). As a result, the patient’s skin turns reddish with spot pimples. The spot pimple forms a ring on the tissue. Candidiasis is a yeast infection that affects delicate parts of the body. Unlike tinea corporis, candidiasis is caused by Candia. The infection site associated with candidiasis includes blood, mouth, throat, genitals and skin. Statistics revealed that women are prone to candidiasis (Clayton & Noble, 2010). As a result, genital candidiasis is prevalent in women. The difference between tinea corporis and candidiasis is determined by the type of fungi and the site of infection.
Characteristics of tinea infections
Tinea corporis can be classified based on the location of infection. As a result, the infection is associated with the scalp, feet, and groin. Tinea capitis is a dermatophyte infection associated with the scalp. The features of tinea capitis include favus, patch, and black dots. Organisms associated with tinea capitis include Trichophyton tonsuruns, and Tirchophyton schoenleinii. Black dots, favus, and gray patch surround the scalp and other tissues of the body. Ringworm infection incude hand, nail and groin. Tinea pedis is contagious and affects all ages. Unlike tinea pedis, the groin infection causes pain and itching. However, the spread depends on the cause of the infection.
Difference between candidiasis and tinea infections
The pathology of tinea corporis is associated with three dermatophytes, which include epidermophyton, trichophyton, and microsporum. As a result, plaques patches are present in the epidermis. Consequently, the hyphae branches after the GMS stain. The pathology of tinea corporis varies among individuals. Consequently, the site of infection influences its development. The infection is stimulated by prolonged treatment and inflammatory changes (Elewski, 1998). As a result, the pathology of the infection differs with the type of ringworm and its location. Compact orthokeratosis, presence of neutrophils, and presence of sandwich sign characterizes the stratum corneum. The pathology of candidiasis is stimulated by Candida albicans. As a result, the predisposing factors include prolonged antibiotic, diabetes mellitus, burns, and frequent exposure. The site of exposure stimulates the pathology of candidiasis. Oral thrush, vulvovaginal, skin, paronychia, and onychomycosis determine the pathology of the infection (Guarner & Brandt, 2011).
Types of tinea infection
The tinea infection is characterized by its location. However, its growth and transmission can be classified in three ways. Anthropophicc, zoophilic, and geophilic organisms influence the spread of the infection. The site of infection is the major classification of tinea infection. As a result, infections based on location include barbae, capitus, corporis, cruris, imbricata, nigra, manuum, pedis, unguium, and versicolor. Unguium affect tissues of the nail while pedis affect the foot. Manuum and nigra infection affect the hand while imbricata affect the body. Cruris affect the groin while capitus affect the head.
Tinea corporis and candidiasis are fungi infections that affect dead tissues. The site of infection determines the type of fungi infection. However, candidiasis is a yeast infection that affects many parts of the body. Unlike tinea corporis, candidiasis is prevalent in women.
Clayton, M. & Noble, C. (2010). Observations on the epidemiology of candida albicans. Journal of Clinical Pathology, 19(9), 76–78.
Elewski, E. (1998). Onychomycosis: Pathogenesis, diagnosis, and management. Clin Microbiol Rev, 11(3), 415–429.
Guarner, J. & Brandt, M. (2011). Histopathologic diagnosis of fungal Infections in the 21st century. Clin Microbiol Rev, 24(2), 247–280.